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Further experimental studies and clinical trials are now necessary to document the relative importance of the oxidant stress caused by an altered redox potential associated with an elevation of homocysteine level, in comparison with alternative molecular mechanisms
Thi gian bn hy t 7-10 pht
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Furthermore, broad-spectrum depalmitoylase inhibitors such as Palmostatin B/M, which covalently target both APT1 and APT2 [178], were initially characterized for their ability to disrupt the S-palmitoylation cycle of oncogenic Ras [179, 180]
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