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A specific gut microbiota dysbiosis of type 2 diabetic mice induces GLP-1 resistance through an enteric NO-dependent and gut-brain axis mechanism Cell Metabolism2017;25(5):1075-1090
EC50 for IR autophosphorylation around 720 nM as a monomer
Patients with certain genetic profiles may benefit from slower dose escalation or the Microdose GLP-1 Protocol, which starts at a lower concentration to minimize early discomfort
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