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In endothelial cell models, BPC-157 significantly enhanced ERK1/2 phosphorylation in a dose-dependent manner, leading to increased cellular proliferation, migration, and vascular tube formation
If tolerance to the satiation effect develops while the dose-limiting nausea does not, the benefitrisk ratio of a given weekly dose would drift over time and a titration schedule built on early-weeks tolerability would be calibrated to the wrong period
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