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In preclinical rodent models, it has demonstrated modulation of adipose tissue and lipid metabolism through indirect mechanisms involving 3-adrenergic receptor pathways, without activating growth hormone or IGF-1-related proliferative effects (Heffernan et al
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This produces downstream effects including sirtuin activation, lipogenesis suppression, and increased energy expenditure
Over time, demyelination may be followed by axonal injury, meaning the nerve fiber itself becomes damaged
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