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Retrieved 19 August 2018 via GlobeNewswire
Similar to disturbances in glutamatergic signaling, preclinical and clinical data point to the primary inhibitory neurotransmitter of the CNS as important for a better understanding of the pathogenesis of ASD and for identifying new therapeutic strategies [117]
the reversible properties of epigenetic modifications posit them as emerging potential targets for next-generation therapeutic interventions [1,2,3,4,5]
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