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Phosphorylated MLCK is ineffective in sustaining the interaction between actin & myosin, and therefore the bronchial smooth muscle relaxes passively (Miller et al, 1983
The principle of the EPR effect is based on the rapid and uncontrolled growth of cancer cells, which enables them to reprogram their vasculature to replenish the increasing demands of oxygen and nutrients, through the formation of structurally abnormal tumor capillaries and blood vessels having defects in their basement membrane with discontinuous epithelium [86, 87]
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