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While early immune responses may aid in the clearance of A and tau aggregates, prolonged activation of microglia and astrocytes leads to chronic inflammation and the release of pro-inflammatory cytokines such as IL-1 and TNF-, promoting neuronal damage in vulnerable brain regions like the neocortex and hippocampus [34,35,36]
Therapeutic prospects for Parkinson disease
This sets precedence for the investigation of T cells and how this may contribute to immunoactivation in TCMR and ABMR
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