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In conclusion, the current study probed the function and mechanism of semaglutide in improving PCOS in vivo experiments, and found that it can be achieved via regulating the PI3K/AKT/mTOR signaling pathway to reduce oxidative stress and autophagy (Figure 9)
At the same time, drug-induced appetite suppression may lead to insufficient intake of protein and key trace elements, further deteriorating the muscle metabolic environment
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