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Jiang F, Ma J, Liang Y, et al

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Mitochondrial ROS production and oxidative mtDNA damage is increased during reoxygenation in the GPX1 knockout mice, in addition to structural abnormalities in cardiac mitochondria and myocytes, suggesting GPX1 may play an important role in protecting cardiac mitochondria from reoxygenation damage in vivo

The coordinated activity of SOD, APX, and CAT establishes a primary detoxification barrier, while the AsAGSH cycle sustains the redox buffering capacity required for continuous ROS neutralization