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Persistent mitochondrial ROS generation leads to lipid peroxidation, increased membrane permeability, mitochondrial uncoupling, and inflammatory signalling via tumor necrosis factor-alpha (TNF-), thereby perpetuating hepatocellular damage [55, 56]

In cultured human cells exposed to heavy metals (Zn, Co, Ni) known to induce DNA breaks and strand instability, Epitalon significantly reduced the extent of genotoxic damage, acting as a protective agent [36]

have shown that DENV-2 infection alters oxidative stress by increasing the levels of malondialdehyde (MDA), the end product of lipid peroxidation, and the oxidized glutathione/GSH ratio 126