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H 2 O 2 produced during the oxidative folding of endoplasmic reticulum proteins has been found to regulate the signal transduction process of nuclear factor E2-related factor 2 (Nrf2), which in turn regulates the expression of downstream glutathione peroxidase 8 (GPX8), which in turn can participate in ER calcium regulation through endoplasmic reticulum calcium ATPase (SERCA) (Figure 7)

Currently, therapies rely largely on pharmacological agents that curb inflammation by targeting immunoregulatory pathways, including Janus Kinase (JAK) inhibitors and other intracellular tyrosine kinases [5, 6]

One review describes its role in accelerating musculoskeletal soft-tissue healing in animal studies