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Currently, synthetic small molecule compounds and natural plant extracts, such as RSL3, terpenoids, and phenols, have been developed to induce ferroptosis for cancer treatment by directly inhibiting GPX4, but these experimental drugs typically exhibit severe cytotoxicity and have unclear pharmacokinetic profiles [47,48,49]

These mechanisms connect to physiological outcomes (faster wound healing, improved tendon repair, protection of the gastrointestinal lining), but current understanding is based largely on laboratory and animal studies, with limited confirmation in controlled human trials

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