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ACSL4 is required to inhibit GPX4-induced ferroptosis, while ALOX12 is dispensable, whereas not required in p53-mediated ferroptosis, and therefore p53 promotes ferroptosis through a GPX4-independent pathway
Iron levels and ferroptosis-related gene expression or mutations should be combined to evaluate which patients are most likely to benefit from treatment targeting ferroptosis
Peptides are naturally occurring molecules made of short chains of amino acids, typically between 2 and 50 amino acids linked together
Notably, many GHK-Cu-regulated genes demonstrate age-dependent dysregulation, with the peptide effectively reversing age-associated expression changes toward more youthful patterns
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