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Lim, J., & Rezaie, A
The mechanism appears to involve improved glucose utilization and metabolic flexibility within the muscle function, allowing for more efficient energy production during sustained activity
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In CKD patients, uremic toxins and ROS probably promote inflammation and oxidative stress through the stimulation of polymorphonuclear lymphocytes, leading to the release of inflammatory cytokines
Mild fluid retention, especially in early weeks of treatment
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