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This constellation of pathology is collectively termed diabetic panvascular disease (DPD), reflecting common molecular mechanisms and interdependent risks among vascular complications
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While iron metabolism and lipid peroxidation trigger ferroptosis under pathological conditions, the precise mechanisms remain incompletely understood and warrant further investigation
Glu, the predominant excitatory neurotransmitter, induces neuronal hyperexcitability through its interaction with ionotropic receptors, such as AMPA, NMDA, and kainate, and metabotropic receptors, known as mGluRs [3]
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