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GLP-1 is part of a communication system between the digestive tract and the brain
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reported that glucagon-induced insulin secretion was inhibited by Exendin 9-39 in purified rat beta-cells ( Nevertheless, the propensity for glucagon to augment insulin secretion through the beta-cell GLP-1R adds another layer of complexity to characterizing the physiological importance of alpha-cell-derived GLP-1 to the incretin effect
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