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A limitation of the current trials is that translation to clinical practice should be made with caution, as is always the case for results obtained in an experimental setting
RANKL expressed on synovial fibroblasts is primarily responsible for bone erosions during joint inflammation
As Attia mentioned, theres a paucity of data so far
Catechins also enhance the activity of endogenous antioxidant enzymes like superoxide dismutase (SOD) and catalase, bolstering the bodys ability to neutralize ROS [210, 211]
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