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for example, multinodular goiter and thyroid adenomas are associated with these deficiencies [1]

In this review, we discuss how the neonatal Fc receptor (FcRn) acts as a regulator of the high levels of not only IgG Abs, but also albumin, by rescuing both these soluble proteins from cellular catabolism, and how a molecular and cellular understanding of this complex biology has spurred an intense interest in the development of FcRn-targeting strategies for the treatment of IgG-driven autoimmune diseases

This conjugate is not excreted as such in the urine but undergoes cleavage, first by the enzyme -glutamyl transpeptidase to release the free glutamyl residue and cysteinyl glycine derivative