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Description

Research models reveal that senescent cells accumulate with aging and in response to various stressors, secreting inflammatory factors known as the senescence-associated secretory phenotype (SASP) that promotes chronic inflammation and can induce senescence in normal cells

A recent publication by Drogue [59, 60], who has extensively investigated the relationship of oxidative stress and aging, has concluded that this event may be in great part be associated with a deficit of cysteine and to a suboptimal intake of SAA