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Disruption of synaptic connectivity and impaired neuronal signaling are early signs of these diseases, and this could be due to defective processing by synaptic mitochondria, which supply the ATP needed as a source of energy for these functions (Petersen et al., 2019)

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To determine whether dysregulated hepatic glutathione homeostasis and TNF can synergistically contribute to hepatocyte death, we performed cell culture studies using both human hepatoma cells and mouse non-transformed hepatocytes

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