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This initial dose is intended to help the body adjust to the medication before increasing the dosage to 0.5 mg, 1 mg, and eventually up to 2.4 mg per week for optimal effects
[1] [2] It works by mimicking the action of the naturally occurring hormone GLP-1, which stimulates insulin secretion in response to food intake, suppresses glucagon release, and slows gastric emptying
The Endocrine Society guideline supports this approach [4]
This review will compare the two modes of signal transduction and propose an evolutionary model of a partnership of the two mechanisms in the eukaryotic cell, with redox-mediated signals being more specific and ancestral and phosphorylative signals being more diffuse but predominant in signal propagation
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