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Several mechanisms to account for the antihypertensive effects associated with SGLT2 inhibitors have been reported and are thought to be mediated partly through enhanced urinary sodium excretion, while glucose-induced osmotic diuresis may cause excessive urine output, thereby contributing to weight loss and a reduction in blood pressure [9]

There is also a natural decrease in glutathione levels with age

Here is the upfront answer: BPC-157 does not directly build muscle the way anabolic steroids, testosterone, or growth hormone secretagogues do

Whether GLP-1/GIP drugs can impact fibrosis remains an open basic and clinical question