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Accordingly, whereas PPAR agonists (fibrates) are classically assumed to improve lipid and cholesterol metabolism via their action on the liver, agonists at PPAR (thiazolidinediones) are assumed to improve insulin sensitivity by enhancing adipose tissue differentiation and fatty acid uptake 10,11,23
These trials must be designed with primary ophthalmic endpoints, such as slowing the rate of RGC loss or preserving visual field, to definitively establish a disease-modifying effect
10.4067/S0718-95162015005000019 68 HernndezI.ChacnO.RodriguezR.PortielesR.LpezY.PujolM.et al
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