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Typical for LCHAD deficiency is the increased risk for female heterozygous carriers to develop hemolysis, elevated liver enzymes, low platelets (HELLP) syndrome and acute fatty liver of pregnancy (AFLP) [135, 136]

The most significant byproduct of this anaerobic shift is the rapid accumulation of lactic acid, leading to prolonged intramuscular acidosis

This approach is often called stacking, and it can help amplify results in areas like muscle growth, fat loss, and recovery

GLP-1 is an incretin hormone produced in the intestinal L-cells that promotes satiety, regulates blood glucose, activates brown fat metabolism and increases energy expenditure (17,18)