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This apparent paradox can be reconciled by conceptualizing ACSL4 as a context-dependent metabolic switch

425 Therefore, for the rejuvenation of the aging thymus, our strategies focused on the FOXN1-TEC axis, such as (1) the enhancement of thymic rejuvenation by the exogenous expression of FoxN1 in TECs via FoxN1 cDNA plasmids and FoxN1 transgenic models

References: Davies MJ, Bajaj HS, Broholm C, et al

This copper-binding motif is critical: the apo-peptide (GHK without copper) shows substantially reduced biological activity in most in vitro assays, indicating that the metal center is required for receptor interaction and downstream signaling