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At the start of this year, we completed a large analysis of the results our members achieved in 2025

5e), but to determine the persistence of agonistreceptor complexes, we adapted our TR-FRET binding assay to include reversible labeling using cleavable SNAP-biotin complexed to streptavidin-terbium to label the surface GLP-1Rs and, after internalization with different FITC-agonists, stripping residual surface receptors of label to ensure that FRET was derived only from internalized receptors (see Fig

A rationally designed CD19 monoclonal antibody-triptolide conjugate for the treatment of systemic lupus erythematosus

M., Proneth, B., and Conrad, M