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Particularly, in fibroblast and neuronal cells from derived from of PKANs patients, lipid peroxidation and alteration of oxidative status (increased ROS production), mitochondrial impairment (including defects in mitochondrial respiration and electrophysiological properties) and premature cell death have been detected [9, 18]
The relationship among GSH depletion, ferroptosis, and senescence has not been reported
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In addition, the chemical structures of different GLP-1 RAs with different pharmacological characteristics from each other
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