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In controlled human malaria infection (CHMI) experiments, a sustained increase in circulating G-MDSC was found in volunteers who developed Plasmodium falciparum parasitemia, which suppressed the proliferation of CD4 + and CD8 + T cells and led to a decrease of lymphocytes, interfering with the production of immune memory against the parasite and suppressing the bodys immune response (256)
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The study was conducted at temperatures ranging from 308 to 338 K and pressures ranging from 12 to 30 MPa
In young mice, it suppressed the release of pro-inflammatory cytokines from macrophages, indicating a potential role in tempering an overactive immune response during youth [44]
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