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Therefore, inhibition or induction of iron decreases can be achieved by overexpression or knockout of NCOA4
In sedentary mice, hepatic FGF-21 mRNA and protein levels were notably elevated compared to active controls, with acute exercise causing a temporary increase [270]
Studies indicate that inhibiting ferroptosis can effectively alleviate IBD-related pathological changes, with protective mechanisms including reduced inflammatory cell infiltration, decreased levels of pro-inflammatory factors, and maintenance of intestinal epithelial barrier integrity
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