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N-acetyl-L-cysteine attenuates oxidative stress-induced bone marrow endothelial cells apoptosis by inhibiting BAX/caspase 3 pathway
It is suggested that activating GIPR can increase the development of white adipose tissue (WAT), which can enhance long-term lipid storage and improve adipocytes ability to remove dietary triglycerides (TAG) [75, 76]
In patients in hyperammonaemic crisis, laboratory testing is required to establish the cause of hyperammonaemia 24
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