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A deposition seems to be a critical pathological trigger in AD and disruption of BBB leads to increased vascular permeability, allowing the entrance and/or hampering the clearance of toxic molecules that can trigger inflammatory and immune responses and, ultimately, neurodegeneration (Iadecola, 2013

Guide for the Care and Use of Laboratory Animals , 8th ed, 220 (National Academies Press, 2011)

GHK-Cu activates fibroblasts, enhancing the production of collagen type I and elastin while promoting matrix metalloproteinases (MMPs) to remodel the extracellular matrix by clearing degraded collagen

One approach is targeting one of the key elements of BAD pathophysiology: the lack of negative feedback inhibition on the bile acid synthesis from the small intestine