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The p38 MAPK became more active, while ERK1/2 and JNK1 activity dropped
Increasing evidence suggests that endometrial receptivity (ER), rather than oocyte quality, may represent the critical limiting factor, particularly in women who exhibit normal embryo development yet experience implantation failure [5, 6]
They constructed an immune-related regulator y axis involving XIST and OIP5-AS1/miR-216a/PIK3CA, which may play a critical role in regulating ferroptosis during AMI progression
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