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In 2014, the GLS inhibitor CB-839, undergoing clinical trials, was reported to possess potent anti-tumor activity, offering a novel avenue for targeted cancer therapy through modulation of glutamine metabolism [14]
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Rhonda Patrick: There's a variety of SNPs that basically make that conversion inefficient
It has also been suggested that the intrinsic control of glucagon secretion in response to high glucose is impaired during the development of diabetes [25]
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