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The spatiotemporal changes in dopamine, neuromelanin and iron characterizing Parkinsons disease
While this is protective against age-related hyperfunction, it theoretically risks blunting the anabolic responses required for new synaptic formation, muscle hypertrophy, or exercise adaptation in subjects who are not already biologically aged
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These alterations greatly decrease its ability to bind to IGF-binding proteins, and it is this property that allows LR3 to stay active in the bloodstream longer than native IGF-1
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