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As early as 1993, it was proposed that Abeta has the ability to form channels within lipid membranes, and this may be its primary toxic effect (Zhaliazka et al
Injections were administered for five consecutive days to the respective experimental groups, starting on the eighth day of the study
The rats treated with BPC 157 exhibited resolved tail spasticity by day 15 post-injury while the rats in the control group did not exhibit sustained spasticity until day 360 [3]
A seed coat-specific -ketoacyl-CoA synthase, KCS12, is critical for preserving seed physical dormancy
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